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See more 7 ghost anime site Fetal exposure to bisphenol A as a risk factor for the development of childhood asthma: An animal model study. Kauppi P. Airway cells, lung-derived immune cells, and blood were obtained from mice in each group. Interestingly, in the more widely used peritoneal sensitization model of allergen immunization, maternal exposure to 0. ECTV infection is characterized by lymphohematogenous spread of the virus from the peripheral site of infection the footpad , and susceptible mice succumb to systemic disease 9. However, these subtle, yet persistent changes due to developmental exposure to BPA did not lead to significant differences in overall airway responsiveness, suggesting that early life exposure to BPA does not exacerbate allergic inflammation into adulthood. The total number of mice in each treatment group is shown at the right. Tilleggsinformasjon Publisert av Top Miners. Gustafsson J. Additional concern arises because BPA is an endocrine disruptor.